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There is now a CONTENT FREEZE for Mercury while we switch to a new platform. It began on Friday, March 10 at 6pm and will end on Wednesday, March 15 at noon. No new content can be created during this time, but all material in the system as of the beginning of the freeze will be migrated to the new platform, including users and groups. Functionally the new site is identical to the old one. webteam@gatech.edu
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Summary Sentence: Shereka Banton and Luke Timmins presenting
Full Summary: The Graduate and Post-Doc (GaP) Seminar Series is a weekly event of research presentations by two graduate students or post-docs conducting bio-related research. The series is organized and sponsored by the Parker H. Petit Institute for Bioengineering and Bioscience (IBB) with additional support from the Wallace H. Coulter Department of Biomedical Engineering. It is held every Wednesday at 12:00pm in IBB 1128 and refreshments are provided. If your research group or department would like to present at future seminars, please contact Manu Platt, PhD.
GaP Seminar Series
Shereka Banton
Luke TimminsShereka Banton - Advisor, Gilda Barabino, PhD
"Exosomal Release of K562-derived MicroRNA: Implications for Sickle Cell Anemia"
Cells have been shown to “communicate” via their exosomal microRNA secretions in vitro. Sickle reticulocyte microRNA expression and possibly function differ compared to RBCs. We are currently investigating whether exosomes secreted by the K562 erythroid progenitor cell line contain microRNAs, and aim to provide a first insight into a possible mechanism of reticulocyte egress from the bone marrow that may be relevant to sickle cell anemia.
Luke Timmins - Advisor, Don Giddens, PhD
"Development of Computational Modeling Techniques to Examine the Association Between Wall Shear Stress and Coronary Allograft Vasculopathy Lesion Formation in Cardiac Transplant Patients"
Coronary allograft vasculopathy (CAV) is the most significant cause of morbidity and mortality for cardiac transplant patients (HTx) after the first year of survival. Furthermore, CAV represents one of the most accelerated forms of occlusive coronary artery disease. We report on the development of patient-specific computational modeling techniques to examine the co-localization of near wall blood flow patterns and CAV lesion formation.
